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Academic Journal

NK cells clear α-synuclein and the depletion of NK cells exacerbates synuclein pathology in a mouse model of α-synucleinopathy.

  • Authors : Earls RH; Department of Physiology and Pharmacology, College of Veterinary Medicine, University of Georgia, Athens, GA 30602.; Menees KB

Subjects: Killer Cells, Natural/Killer Cells, Natural/Killer Cells, Natural/*metabolism ; Synucleinopathies/Synucleinopathies/Synucleinopathies/*metabolism ; Synucleins/Synucleins/Synucleins/*metabolism

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2020 Jan 21; Vol. 117 (3), pp. 1762-1771. Date of Electronic Publication: Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model: Print-Electronic Cited Medium: Internet

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Report

Integrins-A missing link in synuclein's pathogenic mechanism.

  • Authors : Surguchev AA; Department of Surgery, Section of Otolaryngology, Yale School of Medicine, Yale University, New Haven, Connecticut.; Surguchov A

Subjects: Integrins/Integrins/Integrins/*metabolism ; Synucleinopathies/Synucleinopathies/Synucleinopathies/*metabolism ; Synucleins/Synucleins/Synucleins/*metabolism

  • Source: Journal of neuroscience research [J Neurosci Res] 2019 May; Vol. 97 (5), pp. 539-542. Date of Electronic Publication: 2019 Jan 16.Publisher: Wiley Interscience Country of Publication: United States NLM ID: 7600111 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Academic Journal

Synuclein aggregation: When the mouse's away the human will play.

  • Authors : Del Rey NL; Center for Integrative Neuroscience A.C (HM CINAC) and Hospital HM Puerta del Sur, Madrid, Spain.

Subjects: Synucleins* ; alpha-Synuclein*; Animals

  • Source: Movement disorders : official journal of the Movement Disorder Society [Mov Disord] 2016 Aug; Vol. 31 (8), pp. 1143. Date of Electronic Publication: 2016 May 09.Publisher: Wiley-Liss Country of Publication: United States NLM ID: 8610688 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Academic Journal

The chaperone-like activity of α-synuclein attenuates aggregation of its alternatively spliced isoform, 112-synuclein in vitro: plausible cross-talk between isoforms in protein aggregation.

  • Authors : Manda KM; Centre for Academy of Scientific & Innovative Research, CSIR-Indian Institute of Chemical Technology (CSIR-IICT), Hyderabad, Andhra Pradesh, India; Centre for Chemical Biology, CSIR-Indian Institute of Chemical Technology (CSIR-IICT), Hyderabad, Andhra Pradesh, India.

Subjects: Protein Isoforms/Protein Isoforms/Protein Isoforms/*metabolism ; Synucleins/Synucleins/Synucleins/*metabolism ; alpha-Synuclein/alpha-Synuclein/alpha-Synuclein/*metabolism

  • Source: PloS one [PLoS One] 2014 Jun 03; Vol. 9 (6), pp. e98657. Date of Electronic Publication: 2014 Jun 03 (Print Publication: 2014).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet

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Academic Journal

Synucleinopathies from bench to bedside.

Subjects: Lewy Body Disease/Lewy Body Disease/Lewy Body Disease/*metabolism ; Lewy Body Disease/Lewy Body Disease/Lewy Body Disease/*pathology ; Parkinson Disease/Parkinson Disease/Parkinson Disease/*metabolism

  • Source: Parkinsonism & related disorders [Parkinsonism Relat Disord] 2012 Jan; Vol. 18 Suppl 1, pp. S24-7.Publisher: Elsevier Science Country of Publication: England NLM ID: 9513583 Publication Model: Print Cited Medium: Internet ISSN: 1873-5126

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Academic Journal

Synuclein impairs trafficking and signaling of BDNF in a mouse model of Parkinson's disease.

  • Authors : Fang F; Department of Neurology and Institute of Neurology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Department of Neurosciences, University of California San Diego, La Jolla, California, USA.

Subjects: Signal Transduction*; Brain-Derived Neurotrophic Factor/Brain-Derived Neurotrophic Factor/Brain-Derived Neurotrophic Factor/*metabolism ; Parkinson Disease/Parkinson Disease/Parkinson Disease/*metabolism

  • Source: Scientific reports [Sci Rep] 2017 Jun 20; Vol. 7 (1), pp. 3868. Date of Electronic Publication: 2017 Jun 20.Publisher: Nature Publishing Group Country of Publication: England NLM ID: 101563288 Publication Model: Electronic Cited Medium: Internet ISSN:

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Academic Journal

Microglial PHOX and Mac-1 are essential to the enhanced dopaminergic neurodegeneration elicited by A30P and A53T mutant alpha-synuclein.

  • Authors : Zhang W; Neuropharmacology Section, Laboratory of Pharmacology and Chemistry, National Institute of Environmental Health Sciences/National Institutes of Health, Research Triangle Park, North Carolina, USA.; Dallas S

Subjects: Dopamine/Dopamine/Dopamine/*physiology ; Macrophage-1 Antigen/Macrophage-1 Antigen/Macrophage-1 Antigen/*physiology ; Microglia/Microglia/Microglia/*metabolism

  • Source: Glia [Glia] 2007 Aug 15; Vol. 55 (11), pp. 1178-88.Publisher: Wiley-Liss Country of Publication: United States NLM ID: 8806785 Publication Model: Print Cited Medium: Print ISSN: 0894-1491 (Print)

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Academic Journal

Dimerization propensities of Synucleins are not predictive for Synuclein aggregation.

  • Authors : Eckermann K; Department of Neurology, University Medical Center Goettingen, Waldweg 33, 37073 Goettingen, Germany; Cluster of Excellence, Nanoscale Microscopy and Molecular Physiology of the Brain (CNMPB), Goettingen, Germany. Electronic address: .

Subjects: Protein Aggregates* ; Protein Multimerization*; Protein Aggregation, Pathological/Protein Aggregation, Pathological/Protein Aggregation, Pathological/*diagnosis

  • Source: Biochimica et biophysica acta [Biochim Biophys Acta] 2015 Aug; Vol. 1852 (8), pp. 1658-64. Date of Electronic Publication: 2015 May 08.Publisher: Elsevier Pub. Co Country of Publication: Netherlands NLM ID: 0217513 Publication Model: Print-Electronic Cited Medium: Print ISSN:

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Academic Journal

αβγ-Synuclein triple knockout mice reveal age-dependent neuronal dysfunction.

  • Authors : Greten-Harrison B; Program in Cellular Neuroscience, Neurodegeneration, and Repair and Department of Neurology, Yale University, New Haven, CT 06536, USA.; Polydoro M

Subjects: Neurons/Neurons/Neurons/*physiology ; Synapses/Synapses/Synapses/*pathology ; Synaptic Transmission/Synaptic Transmission/Synaptic Transmission/*genetics

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2010 Nov 09; Vol. 107 (45), pp. 19573-8. Date of Electronic Publication: Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model: Print-Electronic Cited Medium: Internet

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Editorial & Opinion

Being too inclusive about synuclein inclusions.

Subjects: Inclusion Bodies/Inclusion Bodies/Inclusion Bodies/*physiology ; Synucleins/Synucleins/Synucleins/*physiology; Humans

  • Source: Nature medicine [Nat Med] 2010 Sep; Vol. 16 (9), pp. 960-1; author reply 961.Publisher: Nature Publishing Company Country of Publication: United States NLM ID: 9502015 Publication Model: Print Cited Medium: Internet ISSN:

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  • 1-10 of  4,182 results for ""alpha-Synuclein""