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Periodical

Novartis pays PTC $1B upfront for midphase Huntington's program, reestablishing itself in R&D race.

Subjects: HUNTINGTON disease; CENTRAL nervous system; PROTEINSHUNTINGTON (W. Va.)NOVARTIS AG 001221845 NOVARTIS AG NOVARTIS AG 001221845 001221845 NOVARTIS AG 001221845 NVS

  • Source: FierceBiotech. 12/2/2024, pN.PAG-N.PAG. 1p.

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Academic Journal

Forkhead box protein p1 is a transcriptional repressor of immune signaling in the CNS: implications for transcriptional dysregulation in Huntington disease.

  • Authors : Tang B; Department of Molecular Biology, The Scripps Research Institute, 10550 North Torrey Pines Rd., La Jolla, CA 92037, USA.; Becanovic K

Subjects: Signal Transduction* ; Transcription, Genetic*; Central Nervous System/Central Nervous System/Central Nervous System/*immunology

  • Source: Human molecular genetics [Hum Mol Genet] 2012 Jul 15; Vol. 21 (14), pp. 3097-111. Date of Electronic Publication: 2012 Apr 06.Publisher: IRL Press at Oxford University Press Country of Publication: England NLM ID: 9208958 Publication Model: Print-Electronic Cited Medium:

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Academic Journal

Synthetic lethal screening in the mammalian central nervous system identifies Gpx6 as a modulator of Huntington's disease.

  • Authors : Shema R; The Broad Institute of MIT and Harvard University, Cambridge, MA 02142; The Picower Institute for Learning and Memory, Cambridge, MA 02139

Subjects: Central Nervous System/Central Nervous System/Central Nervous System/*enzymology ; Central Nervous System/Central Nervous System/Central Nervous System/*pathology ; Glutathione Peroxidase/Glutathione Peroxidase/Glutathione Peroxidase/*metabolism

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2015 Jan 06; Vol. 112 (1), pp. 268-72. Date of Electronic Publication: Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model: Print-Electronic Cited Medium: Internet

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Academic Journal

Treating the whole body in Huntington's disease.

  • Authors : Carroll JB; Behavioral Neuroscience Program, Department of Psychology, Western Washington University, Bellingham, WA, USA. Electronic address: .; Bates GP

Subjects: Central Nervous System/Central Nervous System/Central Nervous System/*pathology ; Huntington Disease/Huntington Disease/Huntington Disease/*pathology ; Huntington Disease/Huntington Disease/Huntington Disease/*therapy

  • Source: The Lancet. Neurology [Lancet Neurol] 2015 Nov; Vol. 14 (11), pp. 1135-42.Publisher: Lancet Pub. Group Country of Publication: England NLM ID: 101139309 Publication Model: Print Cited Medium: Internet ISSN: 1474-4465

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Academic Journal

Management of dysphagia in Huntington's disease: a descriptive review.

Subjects: HUNTINGTON disease; DEGLUTITION disorders; AUTONOMIC nervous system

  • Source: Neurological Sciences. Jun2020, Vol. 41 Issue 6, p1405-1417. 13p. 1 Diagram, 3 Charts.

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Academic Journal

Reduced creatine kinase as a central and peripheral biomarker in Huntington's disease.

  • Authors : Kim J; Geriatric Research Education Clinical Center, New England Veterans Administration VISN 1, Bedford, MA 01730, USA.; Amante DJ

Subjects: Central Nervous System/Central Nervous System/Central Nervous System/*metabolism ; Creatine Kinase, BB Form/Creatine Kinase, BB Form/Creatine Kinase, BB Form/*blood ; Creatine Kinase, BB Form/Creatine Kinase, BB Form/Creatine Kinase, BB Form/*metabolism

  • Source: Biochimica et biophysica acta [Biochim Biophys Acta] 2010 Jul-Aug; Vol. 1802 (7-8), pp. 673-81. Date of Electronic Publication: 2010 May 09.Publisher: Elsevier Pub. Co Country of Publication: Netherlands NLM ID: 0217513 Publication Model: Print-Electronic Cited Medium: Print ISSN:

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Editorial & Opinion

Zinc sulfate could be a potential agent for the treatment of. Huntington's disease through activating central TrkB signaling.

Subjects: Central Nervous System/Central Nervous System/Central Nervous System/*drug effects ; Huntington Disease/Huntington Disease/Huntington Disease/*drug therapy ; Receptor, trkB/Receptor, trkB/Receptor, trkB/*metabolism

  • Source: CNS spectrums [CNS Spectr] 2010 Jan; Vol. 15 (1), pp. 56-7.Publisher: Cambridge University Press Country of Publication: United States NLM ID: 9702877 Publication Model: Print Cited Medium: Print ISSN:

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Academic Journal

Oral administration of the pimelic diphenylamide HDAC inhibitor HDACi 4b is unsuitable for chronic inhibition of HDAC activity in the CNS in vivo.

  • Authors : Beconi M; CHDI Management/CHDI Foundation Inc., Los Angeles, California, United States of America.; Aziz O

Subjects: Central Nervous System/Central Nervous System/Central Nervous System/*metabolism ; Friedreich Ataxia/Friedreich Ataxia/Friedreich Ataxia/*drug therapy ; Histone Deacetylase Inhibitors/Histone Deacetylase Inhibitors/Histone Deacetylase Inhibitors/*pharmacology

  • Source: PloS one [PLoS One] 2012; Vol. 7 (9), pp. e44498. Date of Electronic Publication: 2012 Sep 04.Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: Print-Electronic Cited Medium: Internet

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Academic Journal

Proteomic profiling of plasma in Huntington's disease reveals neuroinflammatory activation and biomarker candidates.

Subjects: Proteomics*; Blood Proteins/Blood Proteins/Blood Proteins/*analysis ; Central Nervous System/Central Nervous System/Central Nervous System/*metabolism

  • Source: Journal of proteome research [J Proteome Res] 2007 Jul; Vol. 6 (7), pp. 2833-40. Date of Electronic Publication: 2007 Jun 07.Publisher: American Chemical Society Country of Publication: United States NLM ID: 101128775 Publication Model: Print-Electronic Cited Medium: Print

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Academic Journal

Reply: More than a co-incidence? Exploring the increased frequency of amyotrophic lateral sclerosis in Huntington disease.

Subjects: *HUNTINGTON disease; *AMYOTROPHIC lateral sclerosis; *EFFERENT pathways

  • Source: Acta Neuropathologica. Feb2023, Vol. 145 Issue 2, p259-261. 3p.

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  • 1-10 of  943 results for ""Huntington Disease""