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Academic Journal

Ketone body 3-hydroxybutyrate elevates cardiac output through peripheral vasorelaxation and enhanced cardiac contractility.

  • Authors : Homilius C; Department of Biomedicine, Aarhus University, Hoegh-Guldbergs Gade 10, 8000, Aarhus, Denmark.; Seefeldt JM

Subjects: Vasodilation* ; Ventricular Function, Left*; Humans

  • Source: Basic research in cardiology [Basic Res Cardiol] 2023 Sep 09; Vol. 118 (1), pp. 37. Date of Electronic Publication: 2023 Sep 09.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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Academic Journal

AMPKα1 deletion in myofibroblasts exacerbates post-myocardial infarction fibrosis by a connexin 43 mechanism.

  • Authors : Dufeys C; Pôle de Recherche Cardiovasculaire (CARD), Institut de Recherche Expérimentale et Clinique (IREC), Université Catholique de Louvain (UCLouvain), 55, Avenue Hippocrate, 1200, Brussels, Belgium.; Daskalopoulos EP

Subjects: Ventricular Function, Left* ; Ventricular Remodeling*; AMP-Activated Protein Kinases/AMP-Activated Protein Kinases/AMP-Activated Protein Kinases/*deficiency

  • Source: Basic research in cardiology [Basic Res Cardiol] 2021 Feb 09; Vol. 116 (1), pp. 10. Date of Electronic Publication: 2021 Feb 09.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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Academic Journal

Vascular conditioning prevents adverse left ventricular remodelling after acute myocardial infarction: a randomised remote conditioning study.

  • Authors : Ikonomidis I; 2nd Department of Cardiology, Medical School, Attikon Hospital, National and Kapodistrian University of Athens, Rimini 1, Haidari, 12462, Athens, Greece. .; Vlastos D

Subjects: Ischemic Postconditioning*/Ischemic Postconditioning*/Ischemic Postconditioning*/adverse effects ; Percutaneous Coronary Intervention*/Percutaneous Coronary Intervention*/Percutaneous Coronary Intervention*/adverse effects ; Ventricular Function, Left*

  • Source: Basic research in cardiology [Basic Res Cardiol] 2021 Feb 06; Vol. 116 (1), pp. 9. Date of Electronic Publication: 2021 Feb 06.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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Academic Journal

Intracellular O-linked glycosylation directly regulates cardiomyocyte L-type Ca 2+ channel activity and excitation-contraction coupling.

  • Authors : Ednie AR; Department of Neuroscience, Cell Biology and Physiology, Boonshoft School of Medicine and College of Science and Mathematics, Wright State University, 143 Biological Sciences II, 3640 Colonel Glenn Hwy, Dayton, OH, 45435, USA. .; Bennett ES

Subjects: Excitation Contraction Coupling*/Excitation Contraction Coupling*/Excitation Contraction Coupling*/drug effects ; Ventricular Function, Left*/Ventricular Function, Left*/Ventricular Function, Left*/drug effects; Calcium Channels, L-Type/Calcium Channels, L-Type/Calcium Channels, L-Type/*metabolism

  • Source: Basic research in cardiology [Basic Res Cardiol] 2020 Sep 10; Vol. 115 (6), pp. 59. Date of Electronic Publication: 2020 Sep 10.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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Academic Journal

Deletion of obscurin immunoglobulin domains Ig58/59 leads to age-dependent cardiac remodeling and arrhythmia.

  • Authors : Grogan A; Department of Biochemistry and Molecular Biology, University of Maryland School of Medicine, Baltimore, MD, 21201, USA.; Coleman A

Subjects: Heart Rate* ; Ventricular Function, Left* ; Ventricular Remodeling*

  • Source: Basic research in cardiology [Basic Res Cardiol] 2020 Sep 10; Vol. 115 (6), pp. 60. Date of Electronic Publication: 2020 Sep 10.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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Academic Journal

M1-like macrophage-derived exosomes suppress angiogenesis and exacerbate cardiac dysfunction in a myocardial infarction microenvironment.

  • Authors : Liu S; Guangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China. .; Chen J

Subjects: Cellular Microenvironment* ; Neovascularization, Physiologic* ; Ventricular Function, Left*

  • Source: Basic research in cardiology [Basic Res Cardiol] 2020 Feb 28; Vol. 115 (2), pp. 22. Date of Electronic Publication: 2020 Feb 28.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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Academic Journal

Triad3A attenuates pathological cardiac hypertrophy involving the augmentation of ubiquitination-mediated degradation of TLR4 and TLR9.

  • Authors : Lu X; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China.; He Y

Subjects: Ventricular Function, Left* ; Ventricular Remodeling*; Hypertrophy, Left Ventricular/Hypertrophy, Left Ventricular/Hypertrophy, Left Ventricular/*prevention & control

  • Source: Basic research in cardiology [Basic Res Cardiol] 2020 Feb 01; Vol. 115 (2), pp. 19. Date of Electronic Publication: 2020 Feb 01.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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Academic Journal

Selective intrarenal delivery of mesenchymal stem cell-derived extracellular vesicles attenuates myocardial injury in experimental metabolic renovascular disease.

  • Authors : Zhang L; Division of Nephrology and Hypertension, Mayo Clinic, Rochester, MN, 55905, USA.; Department of Urology, Zhongda Hospital, Southeast University, Nanjing, 210009, People's Republic of China.

Subjects: Mesenchymal Stem Cell Transplantation* ; Ventricular Function, Left* ; Ventricular Remodeling*

  • Source: Basic research in cardiology [Basic Res Cardiol] 2020 Jan 14; Vol. 115 (2), pp. 16. Date of Electronic Publication: 2020 Jan 14.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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Academic Journal

E2f1 deletion attenuates infarct-induced ventricular remodeling without affecting O-GlcNAcylation.

  • Authors : Dassanayaka S; Division of Cardiovascular Medicine, Department of Medicine, Diabetes and Obesity Center, Institute of Molecular Cardiology, University of Louisville, 580 South Preston Street-321F, Delia Baxter Building-321F, Louisville, KY, 40202, USA.; Brittian KR

Subjects: Ventricular Function, Left* ; Ventricular Remodeling*; E2F1 Transcription Factor/E2F1 Transcription Factor/E2F1 Transcription Factor/*deficiency

  • Source: Basic research in cardiology [Basic Res Cardiol] 2019 May 31; Vol. 114 (4), pp. 28. Date of Electronic Publication: 2019 May 31.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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Academic Journal

Raf kinase inhibitor protein mediates myocardial fibrosis under conditions of enhanced myocardial oxidative stress.

  • Authors : Kazakov A; Klinik für Innere Medizin III, Kardiologie, Angiologie und Internistische Intensivmedizin, Universität/Universitätsklinikum des Saarlandes, Kirrberger Strasse 100, IMED, 66421, Homburg, Germany. .; Hall RA

Subjects: Oxidative Stress* ; Ventricular Function, Left* ; Ventricular Remodeling*/Ventricular Remodeling*/Ventricular Remodeling*/genetics

  • Source: Basic research in cardiology [Basic Res Cardiol] 2018 Sep 06; Vol. 113 (6), pp. 42. Date of Electronic Publication: 2018 Sep 06.Publisher: Steinkopff Country of Publication: Germany NLM ID: 0360342 Publication Model: Electronic Cited Medium:

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  • 1-10 of  382 results for ""Ventricular Function, Left""