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Academic Journal

Posttranslational modifications by ADAM10 shape myeloid antigen-presenting cell homeostasis in the splenic marginal zone.

  • Authors : Diener N; Institute for Molecular Medicine, Paul Klein Center for Immune Intervention, University Medical Center of the Johannes Gutenberg-University, 55131 Mainz, Germany.; Fontaine JF

Subjects: ADAM10 Protein/ADAM10 Protein/ADAM10 Protein/*metabolism ; Amyloid Precursor Protein Secretases/Amyloid Precursor Protein Secretases/Amyloid Precursor Protein Secretases/*metabolism ; Dendritic Cells/Dendritic Cells/Dendritic Cells/*metabolism

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2021 Sep 21; Vol. 118 (38).Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model: Print Cited

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Academic Journal

Cell-autonomous FLT3L shedding via ADAM10 mediates conventional dendritic cell development in mouse spleen.

  • Authors : Fujita K; Department of Dentistry and Oral Surgery, Keio University School of Medicine, Tokyo 160-8582, Japan.; Department of Dermatology, Keio University School of Medicine, Tokyo 160-8582, Japan.

Subjects: ADAM10 Protein/ADAM10 Protein/ADAM10 Protein/*metabolism ; Amyloid Precursor Protein Secretases/Amyloid Precursor Protein Secretases/Amyloid Precursor Protein Secretases/*metabolism ; Cell Differentiation/Cell Differentiation/Cell Differentiation/*immunology

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2019 Jul 16; Vol. 116 (29), pp. 14714-14723. Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model:

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Academic Journal

Regulation of adipose tissue inflammation by interleukin 6.

  • Authors : Han MS; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA 01605.; White A

Subjects: Adipose Tissue/Adipose Tissue/Adipose Tissue/*immunology ; Interleukin-6/Interleukin-6/Interleukin-6/*immunology ; Obesity/Obesity/Obesity/*immunology

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2020 Feb 11; Vol. 117 (6), pp. 2751-2760. DatePublisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model:

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Academic Journal

Nicastrin functions to sterically hinder γ-secretase- substrate interactions driven by substrate transmembrane domain.

Subjects: NICASTRIN; SECRETASES; ALZHEIMER'S disease

  • Source: Proceedings of the National Academy of Sciences of the United States of America; 2/2/2016, Vol. 113 Issue 5, pE509-E518, 10p

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Academic Journal

Oral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humans.

Subjects: DEMENTIA; ALZHEIMER'S disease; METABOLIC syndrome

  • Source: Proceedings of the National Academy of Sciences of the United States of America; 4/1/2014, Vol. 111 Issue 13, p4940-4945, 6p

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Academic Journal

M344 promotes nonamyloidogenic amyloid precursor protein processing while normalizing Alzheimer's disease genes and improving memory.

  • Authors : Volmar CH; Center for Therapeutic Innovation, Miller School of Medicine, University of Miami, Miami, FL 33136; .

Subjects: Alzheimer Disease/Alzheimer Disease/Alzheimer Disease/*drug therapy ; Amyloid beta-Peptides/Amyloid beta-Peptides/Amyloid beta-Peptides/*metabolism ; Histone Deacetylase Inhibitors/Histone Deacetylase Inhibitors/Histone Deacetylase Inhibitors/*pharmacology

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2017 Oct 24; Vol. 114 (43), pp. E9135-E9144. Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model:

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Academic Journal

The adherens junctions control susceptibility to Staphylococcus aureus α-toxin.

  • Authors : Popov LM; Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, CA 94305;

Subjects: Adherens Junctions/Adherens Junctions/Adherens Junctions/*metabolism ; Bacterial Toxins/Bacterial Toxins/Bacterial Toxins/*metabolism ; Hemolysin Proteins/Hemolysin Proteins/Hemolysin Proteins/*metabolism

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2015 Nov 17; Vol. 112 (46), pp. 14337-42. DatePublisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model:

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Academic Journal

Activation of peroxisome proliferator-activated receptor α stimulates ADAM10-mediated proteolysis of APP.

  • Authors : Corbett GT; Graduate Program in Neuroscience, Department of Neurological Sciences, Rush University Medical Center, Chicago, IL 60612; Department of Neurological Sciences, Rush University Medical Center, Chicago, IL 60612

Subjects: ADAM Proteins/ADAM Proteins/ADAM Proteins/*metabolism ; Alzheimer Disease/Alzheimer Disease/Alzheimer Disease/*metabolism ; Amyloid Precursor Protein Secretases/Amyloid Precursor Protein Secretases/Amyloid Precursor Protein Secretases/*metabolism

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2015 Jul 07; Vol. 112 (27), pp. 8445-50. Date Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model:

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Academic Journal

Membrane-enabled dimerization of the intrinsically disordered cytoplasmic domain of ADAM10.

  • Authors : Deng W; Aflac Cancer and Blood Disorders Center, Department of Pediatrics, Emory University School of Medicine, Atlanta, GA 30322; and.

Subjects: ADAM Proteins/ADAM Proteins/ADAM Proteins/*metabolism ; Amyloid Precursor Protein Secretases/Amyloid Precursor Protein Secretases/Amyloid Precursor Protein Secretases/*metabolism ; Cell Membrane/Cell Membrane/Cell Membrane/*metabolism

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2014 Nov 11; Vol. 111 (45), pp. 15987-92. DatePublisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model:

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Academic Journal

Oral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humans.

  • Authors : Cai W; Department of Geriatrics, Division of Experimental Diabetes, Medicine, Human Genetics, and Psychiatry, The Icahn School of Medicine at Mount Sinai, New York, NY 10029.; Uribarri J

Subjects: Dementia/Dementia/Dementia/*pathology ; Glycation End Products, Advanced/Glycation End Products, Advanced/Glycation End Products, Advanced/*adverse effects ; Metabolic Syndrome/Metabolic Syndrome/Metabolic Syndrome/*pathology

  • Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2014 Apr 01; Vol. 111 (13), pp. 4940-5. Date Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model:

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  • 1-10 of  15 results for ""ADAM10 Protein""