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Bacterial pathogens activate a common inflammatory pathway through IFNλ regulation of PDCD4.

• Authors : Cohen TS; Department of Pediatrics, Columbia University, New York, New York, United States of America.; Prince AS

Subjects: Apoptosis Regulatory Proteins/Apoptosis Regulatory Proteins/Apoptosis Regulatory Proteins/*biosynthesis ; Cytokines/Cytokines/Cytokines/*metabolism ; Pneumonia, Staphylococcal/Pneumonia, Staphylococcal/Pneumonia, Staphylococcal/*metabolism

• Source: PLoS pathogens [PLoS Pathog] 2013; Vol. 9 (10), pp. e1003682. Date of Electronic Publication: 2013 Oct 03.Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: Print-Electronic Cited Medium: Internet

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A specific innate immune response silences the virulence of Pseudomonas aeruginosa in a latent infection model in the Drosophila melanogaster host.

• Authors : Chen J; Sino-French Hoffmann Institute, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China.; Lin G

Subjects: Drosophila melanogaster*/Drosophila melanogaster*/Drosophila melanogaster*/microbiology ; Drosophila melanogaster*/Drosophila melanogaster*/Drosophila melanogaster*/immunology ; Pseudomonas aeruginosa*/Pseudomonas aeruginosa*/Pseudomonas aeruginosa*/pathogenicity

• Source: PLoS pathogens [PLoS Pathog] 2024 Jun 04; Vol. 20 (6), pp. e1012252. Date of Electronic Publication: 2024 Jun 04 (Print Publication: 2024).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet

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Pseudomonas aeruginosa senses and responds to epithelial potassium flux via Kdp operon to promote biofilm.

• Authors : Rapsinski GJ; Department of Microbiology and Molecular Genetics, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, United State of America.; Division of Infectious Disease, Department of Pediatrics, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, United States of America.

Subjects: Biofilms*/Biofilms*/Biofilms*/growth & development ; Pseudomonas aeruginosa*/Pseudomonas aeruginosa*/Pseudomonas aeruginosa*/genetics ; Pseudomonas aeruginosa*/Pseudomonas aeruginosa*/Pseudomonas aeruginosa*/metabolism

• Source: PLoS pathogens [PLoS Pathog] 2024 May 31; Vol. 20 (5), pp. e1011453. Date of Electronic Publication: 2024 May 31 (Print Publication: 2024).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet

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Pseudomonas aeruginosa two-component system CprRS regulates HigBA expression and bacterial cytotoxicity in response to LL-37 stress.

• Authors : Song Y; College of Life Science, Sichuan Normal University, Chengdu, China.; Zhang S

Subjects: Pseudomonas aeruginosa*/Pseudomonas aeruginosa*/Pseudomonas aeruginosa*/metabolism ; Pseudomonas Infections*/Pseudomonas Infections*/Pseudomonas Infections*/genetics ; Pseudomonas Infections*/Pseudomonas Infections*/Pseudomonas Infections*/microbiology

• Source: PLoS pathogens [PLoS Pathog] 2024 Jan 10; Vol. 20 (1), pp. e1011946. Date of Electronic Publication: 2024 Jan 10 (Print Publication: 2024).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet

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Pseudomonas aeruginosa pulmonary infection results in S100A8/A9-dependent cardiac dysfunction.

• Authors : Kumar N; Department of Microbial Infection and Immunity, College of Medicine, The Ohio State University, Wexner Medical Center, Columbus, Ohio, United States of America.; Pestrak MJ

Subjects: Pseudomonas Infections* ; Heart Diseases*; Animals

• Source: PLoS pathogens [PLoS Pathog] 2023 Aug 25; Vol. 19 (8), pp. e1011573. Date of Electronic Publication: 2023 Aug 25 (Print Publication: 2023).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet

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A compensatory RNase E variation increases Iron Piracy and Virulence in multidrug-resistant Pseudomonas aeruginosa during Macrophage infection.

• Authors : Vaillancourt M; Department of Pathology and Laboratory Medicine, Cedars-Sinai Medical Center, Los Angeles, California, United States of America.; Galdino ACM

Subjects: Iron*/Iron*/Iron*/metabolism ; Pseudomonas Infections*/Pseudomonas Infections*/Pseudomonas Infections*/drug therapy ; Pseudomonas Infections*/Pseudomonas Infections*/Pseudomonas Infections*/metabolism

• Source: PLoS pathogens [PLoS Pathog] 2023 Apr 07; Vol. 19 (4), pp. e1010942. Date of Electronic Publication: 2023 Apr 07 (Print Publication: 2023).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet

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Current concepts on Pseudomonas aeruginosa interaction with human airway epithelium.

• Authors : Muggeo A; Université de Reims Champagne-Ardenne, INSERM, CHU de Reims, Laboratoire de bactériologie-Virologie-Hygiène hospitalière-Parasitologie-Mycologie, P3Cell, U 1250, Reims, France.; Coraux C

Subjects: Pseudomonas aeruginosa* ; Pseudomonas Infections*/Pseudomonas Infections*/Pseudomonas Infections*/microbiology; Humans

• Source: PLoS pathogens [PLoS Pathog] 2023 Mar 30; Vol. 19 (3), pp. e1011221. Date of Electronic Publication: 2023 Mar 30 (Print Publication: 2023).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet

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The opportunistic pathogen Pseudomonas aeruginosa exploits bacterial biotin synthesis pathway to benefit its infectivity.

• Authors : Shi Y; Department of Microbiology, and Department of General Intensive Care Unit of the Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, Zhejiang, China.; Cao Q

Subjects: Biotin*/Biotin*/Biotin*/chemistry ; Biotin*/Biotin*/Biotin*/metabolism ; Pseudomonas Infections*

• Source: PLoS pathogens [PLoS Pathog] 2023 Jan 23; Vol. 19 (1), pp. e1011110. Date of Electronic Publication: 2023 Jan 23 (Print Publication: 2023).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet

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Pathogen to commensal? Longitudinal within-host population dynamics, evolution, and adaptation during a chronic >16-year Burkholderia pseudomallei infection.

• Authors : Pearson, Talima; Sahl, Jason W.; Hepp, Crystal M.

• Source: PLoS Pathogens. March 5, 2020, Vol. 16 Issue 3, e1008298.

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Rab32 GTPase, as a direct target of miR-30b/c, controls the intracellular survival of Burkholderia pseudomallei by regulating phagosome maturation.

• Authors : Hu, Zhi-qiang; Rao, Cheng-long; Tang, Meng-ling

• Source: PLoS Pathogens. June 14, 2019, Vol. 15 Issue 6, e1007879.

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