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Academic Journal

Genetic inhibition of nuclear factor of activated T-cell c2 prevents atrial fibrillation in CREM transgenic mice.

  • Authors : Ni L; Division of Cardiology, Department of Internal Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Jiefang Avenue NO.1095, Qiaokou District, Wuhan, 430030, China.; Hubei Key Laboratory of Genetics and Molecular Mechanisms of Cardiological Disorders, Jiefang Avenue NO.1095, Qiaokou District, Wuhan, 430030, China.

Subjects: Atrial Fibrillation*/Atrial Fibrillation*/Atrial Fibrillation*/genetics ; Atrial Fibrillation*/Atrial Fibrillation*/Atrial Fibrillation*/prevention & control ; Atrial Fibrillation*/Atrial Fibrillation*/Atrial Fibrillation*/pathology

  • Source: Cardiovascular research [Cardiovasc Res] 2022 Oct 21; Vol. 118 (13), pp. 2805-2818.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print Cited Medium: Internet ISSN: 1755-3245

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Academic Journal

Regulation of protease-activated receptor-1 by vasodilatory prostaglandins via NFAT.

  • Authors : Rosenkranz AC; Institut für Pharmakologie und Klinische Pharmakologie, Heinrich-Heine-Universität Düsseldorf, Universitätsstrasse 1, 40225 Düsseldorf, Germany.; Rauch BH

Subjects: Myocytes, Smooth Muscle/Myocytes, Smooth Muscle/Myocytes, Smooth Muscle/*metabolism ; NFATC Transcription Factors/NFATC Transcription Factors/NFATC Transcription Factors/*metabolism ; Prostaglandins/Prostaglandins/Prostaglandins/*metabolism

  • Source: Cardiovascular research [Cardiovasc Res] 2009 Sep 01; Vol. 83 (4), pp. 778-84. Date of Electronic Publication: 2009 May 21.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Academic Journal

Enhanced expression of DYRK1A in cardiomyocytes inhibits acute NFAT activation but does not prevent hypertrophy in vivo.

  • Authors : Grebe C; Department of Cardiology and Pulmonology, Georg-August-University, Robert-Koch Str. 40, 37075 Göttingen, Germany.; Klingebiel TM

Subjects: Cardiomegaly/Cardiomegaly/Cardiomegaly/*genetics ; Cardiomegaly/Cardiomegaly/Cardiomegaly/*metabolism ; Myocytes, Cardiac/Myocytes, Cardiac/Myocytes, Cardiac/*metabolism

  • Source: Cardiovascular research [Cardiovasc Res] 2011 Jun 01; Vol. 90 (3), pp. 521-8. Date of Electronic Publication: 2011 Jan 27.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Academic Journal

Proteasome functional insufficiency activates the calcineurin-NFAT pathway in cardiomyocytes and promotes maladaptive remodelling of stressed mouse hearts.

  • Authors : Tang M; Division of Basic Biomedical Sciences, Sanford School of Medicine of the University of South Dakota, 414 East Clark Street, Lee Medical Building, Vermillion, SD 57069, USA.; Li J

Subjects: Calcineurin/Calcineurin/Calcineurin/*physiology ; Heart Failure/Heart Failure/Heart Failure/*physiopathology ; Myocytes, Cardiac/Myocytes, Cardiac/Myocytes, Cardiac/*physiology

  • Source: Cardiovascular research [Cardiovasc Res] 2010 Dec 01; Vol. 88 (3), pp. 424-33. Date of Electronic Publication: 2010 Jul 02.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Academic Journal

A gene therapeutic approach to inhibit calcium and integrin binding protein 1 ameliorates maladaptive remodelling in pressure overload.

  • Authors : Grund A; Klinik für Kardiologie und Angiologie, Medizinische Hochschule Hannover, Carl-Neuberg-Strasse 1, Hannover, Germany.; Szaroszyk M

Subjects: RNAi Therapeutics* ; Ventricular Function, Left* ; Ventricular Remodeling*

  • Source: Cardiovascular research [Cardiovasc Res] 2019 Jan 01; Vol. 115 (1), pp. 71-82.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print Cited Medium: Internet ISSN: 1755-3245

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Academic Journal

A novel method for high precision aortic constriction that allows for generation of specific cardiac phenotypes in mice.

  • Authors : Melleby AO; Institute for Experimental Medical Research, Oslo University Hospital and University of Oslo, Norway.; KG Jebsen Center for Cardiac Research, University of Oslo, Oslo, Norway.

Subjects: Myocardium*/Myocardium*/Myocardium*/metabolism ; Myocardium*/Myocardium*/Myocardium*/pathology ; Ventricular Function, Left*

  • Source: Cardiovascular research [Cardiovasc Res] 2018 Oct 01; Vol. 114 (12), pp. 1680-1690.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print Cited Medium: Internet ISSN: 1755-3245

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Academic Journal

Loss of AKAP150 promotes pathological remodelling and heart failure propensity by disrupting calcium cycling and contractile reserve.

  • Authors : Li L; Department of Physiology and Biophysics, University of Washington, 1705 NE Pacific Street, Seattle, WA 98195, USA.; Li J

Subjects: Calcium Signaling* ; Ventricular Function* ; Ventricular Remodeling*

  • Source: Cardiovascular research [Cardiovasc Res] 2017 Feb; Vol. 113 (2), pp. 147-159. Date of Electronic Publication: 2016 Nov 17.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Academic Journal

Caveolae-localized L-type Ca2+ channels do not contribute to function or hypertrophic signalling in the mouse heart.

  • Authors : Correll RN; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, 240 Albert Sabin Way, Cincinnati, OH 45229, USA.; Makarewich CA

Subjects: Calcium Signaling* ; Ventricular Function, Left*; Calcium Channels, L-Type/Calcium Channels, L-Type/Calcium Channels, L-Type/*metabolism

  • Source: Cardiovascular research [Cardiovasc Res] 2017 Jun 01; Vol. 113 (7), pp. 749-759.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print Cited Medium: Internet ISSN: 1755-3245

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Academic Journal

Phosphoinositide 3-kinase gamma controls inflammation-induced myocardial depression via sequential cAMP and iNOS signalling.

  • Authors : Ndongson-Dongmo B; Institute of Molecular Cell Biology, Jena University Hospital, Friedrich Schiller University, Hans-Knöll-Straße 2, D-07745 Jena, Germany Integrated Research and Treatment Center, Center for Sepsis Control and Care, Jena University Hospital, Jena, Germany.; Heller R

Subjects: Cardiomyopathies/Cardiomyopathies/Cardiomyopathies/*enzymology ; Class Ib Phosphatidylinositol 3-Kinase/Class Ib Phosphatidylinositol 3-Kinase/Class Ib Phosphatidylinositol 3-Kinase/*metabolism ; Cyclic AMP/Cyclic AMP/Cyclic AMP/*metabolism

  • Source: Cardiovascular research [Cardiovasc Res] 2015 Nov 01; Vol. 108 (2), pp. 243-53. Date of Electronic Publication: 2015 Sep 02.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Academic Journal

Pathogenesis of hypertrophic cardiomyopathy caused by myozenin 2 mutations is independent of calcineurin activity.

  • Authors : Ruggiero A; Center for Cardiovascular Genetics, The Brown Foundation Institute of Molecular Medicine, The University of Texas Health Science Center and Texas Heart Institute, 6770 Bertner Street, Suite C900A, Houston, TX 77030, USA.; Chen SN

Subjects: Mutation*; Calcineurin/Calcineurin/Calcineurin/*metabolism ; Cardiomyopathy, Hypertrophic/Cardiomyopathy, Hypertrophic/Cardiomyopathy, Hypertrophic/*genetics

  • Source: Cardiovascular research [Cardiovasc Res] 2013 Jan 01; Vol. 97 (1), pp. 44-54. Date of Electronic Publication: 2012 Sep 17.Publisher: Oxford Journals Country of Publication: England NLM ID: 0077427 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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  • 1-10 of  30 results for ""NFATC Transcription Factors""