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NT69L blocks ethanol-induced increase of dopamine and glutamate levels in striatum of mouse
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- Author(s): Li, Zhimin ; Boules, Mona1; Richelson, Elliott1
- Source:
Neuroscience Letters. Jan2011, Vol. 487 Issue 3, p322-324. 3p.- Subject Terms:
- Source:
- Additional Information
- Abstract: Abstract: Recent study shows that NT69L, an analog of neurotensin (NT) (8–13), reduces ethanol consumption and preference in mice through modulation of neurotensin receptor subtype one. The current study showed that NT69L significantly decreased ethanol-induced increase of dopamine and glutamate levels in striatum of mouse. These data suggest that NT69L prevents ethanol consumption through the modulation of both dopaminergic and glutamatergic systems implicated in ethanol addiction. NT agonists may provide novel treatment for alcohol addiction. [Copyright &y& Elsevier]
- Abstract: Copyright of Neuroscience Letters is the property of Elsevier B.V. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)
- Abstract:
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