A Child's Nutrition and Epigenetics.

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  • Author(s): Silao CLT;Silao CLT;Silao CLT
  • Source:
    Nestle Nutrition Institute workshop series [Nestle Nutr Inst Workshop Ser] 2023; Vol. 97, pp. 1-10. Date of Electronic Publication: 2023 Apr 06.
  • Publication Type:
    Journal Article; Review
  • Language:
    English
  • Additional Information
    • Source:
      Publisher: Karger Country of Publication: Switzerland NLM ID: 101577268 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1664-2155 (Electronic) Linking ISSN: 16642147 NLM ISO Abbreviation: Nestle Nutr Inst Workshop Ser Subsets: MEDLINE
    • Publication Information:
      Original Publication: Basel : Karger
    • Subject Terms:
      Chronic Disease* ; Diet* ; Epigenesis, Genetic* ; Infant Nutritional Physiological Phenomena*; Humans ; Nutritional Status ; Infant
    • Abstract:
      Studies have shown a dramatic increase in the incidence and the prevalence of chronic diseases such as type 2 diabetes mellitus and cardiovascular disorders over the last several decades. Environmental triggers and nutrition are considered major contributors to this increase. The first 1,000 days of life, which is the period between conception and the first 2 years of age, is considered the time for environmental factors, such as nutrition, to exert their positive and most crucial effects on a child's health. Nutrigenomics, the study of how genes and food components interact, looks into diet-altering disease development by modulating processes involved with the onset, progression, and severity of disease. These factors affecting the development of these chronic diseases are thought to be mediated by epigenetic mechanisms, which are heritable and reversible, and carry genetic information without changing the nucleotide sequence of the genome and are also mediated by maternal and postnatal nutrition.
      (© 2023 S. Karger AG, Basel.)
    • Publication Date:
      Date Created: 20230406 Date Completed: 20230410 Latest Revision: 20230708
    • Publication Date:
      20240829
    • Accession Number:
      10.1159/000528991
    • Accession Number:
      37023732