New Findings from Auburn University in the Area of Exercise Therapy Reported (Skeletal Muscle Myosin Heavy Chain Fragmentation As a Potential Marker of Protein Degradation In Response To Resistance Training and Disuse Atrophy).

A study conducted at Auburn University in Alabama examined the effects of resistance exercise, cycling exercise, and disuse atrophy on myosin heavy chain (MyHC) protein fragmentation. The study found that resistance exercise and disuse atrophy both increased MyHC protein fragmentation. The researchers suggest that further research is needed to understand how aging and disease-associated muscle atrophy impact these outcomes. The study was funded by various organizations, including the National Institutes of Health in the United States. [Extracted from the article]

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