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Closed for Staff Day
Phone: (843) 766-6635
Wando Mount Pleasant Library
Closed for Staff Day
Phone: (843) 805-6888
Village Library
Closed for Staff Day
Phone: (843) 884-9741
St. Paul's/Hollywood Library
Closed for Staff Day
Phone: (843) 889-3300
Otranto Road Library
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Phone: (843) 572-4094
Mt. Pleasant Library
Closed for Staff Day
Phone: (843) 849-6161
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Phone: (843) 552-6466
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Phone: (843) 722-7550
Baxter-Patrick James Island
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Closed for Staff Day
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Spatiotemporal development and the regulatory mechanisms of cardiac resident macrophages: Contribution in cardiac development and steady state.
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- Author(s): Qin, Demeng (AUTHOR); Zhang, Ying (AUTHOR); Liu, Fang (AUTHOR); Xu, Xiang (AUTHOR); Jiang, Haiqiang (AUTHOR); Su, Zhaoliang (AUTHOR); Xia, Lin (AUTHOR)
- Source:
Acta Physiologica. Mar2024, Vol. 240 Issue 3, p1-16. 16p. - Source:
- Additional Information
- Subject Terms:
- Abstract: Cardiac resident macrophages (CRMs) are integral components of the heart and play significant roles in cardiac development, steady‐state, and injury. Advances in sequencing technology have revealed that CRMs are a highly heterogeneous population, with significant differences in phenotype and function at different developmental stages and locations within the heart. In addition to research focused on diseases, recent years have witnessed a heightened interest in elucidating the involvement of CRMs in heart development and the maintenance of cardiac function. In this review, we primarily concentrated on summarizing the developmental trajectories, both spatial and temporal, of CRMs and their impact on cardiac development and steady‐state. Moreover, we discuss the possible factors by which the cardiac microenvironment regulates macrophages from the perspectives of migration, proliferation, and differentiation under physiological conditions. Gaining insight into the spatiotemporal heterogeneity and regulatory mechanisms of CRMs is of paramount importance in comprehending the involvement of macrophages in cardiac development, injury, and repair, and also provides new ideas and therapeutic methods for treating heart diseases. [ABSTRACT FROM AUTHOR]
- Abstract: Copyright of Acta Physiologica is the property of Wiley-Blackwell and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)
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