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West Ashley Library
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Phone: (843) 766-6635
Folly Beach Library
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Phone: (843) 588-2001
Edgar Allan Poe/Sullivan's Island Library
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Wando Mount Pleasant Library
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Phone: (843) 805-6888
Village Library
9 a.m. - 1 p.m.
Phone: (843) 884-9741
St. Paul's/Hollywood Library
9 a.m. - 8 p.m.
Phone: (843) 889-3300
Otranto Road Library
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McClellanville Library
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Keith Summey North Charleston Library
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Hurd/St. Andrews Library
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Dorchester Road Library
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Phone: (843) 722-7550
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Phone: (843) 805-6930
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Eradication of Helicobacter pylori infection decreases risk for dyslipidemia: A cohort study.
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- Author(s): Park, Yewan; Kim, Tae Jun; Lee, Hyuk; Yoo, Heejin; Sohn, Insuk; Min, Yang Won; Min, Byung‐Hoon; Lee, Jun Haeng; Rhee, Poong‐Lyul; Kim, Jae J.
- Source:
Helicobacter; Apr2021, Vol. 26 Issue 2, p1-9, 9p- Subject Terms:
- Source:
- Additional Information
- Abstract: Background: Previous studies have suggested a relationship between Helicobacter pylori infection and dyslipidemia; however, large‐scale longitudinal studies have not elucidated this association. This study assessed the longitudinal effects of H. pylori infection and eradication on lipid profiles in a large cohort. Methods: This cohort study included 2,626 adults without dyslipidemia at baseline, who participated in a repeated, regular health‐screening examination, which included upper gastrointestinal endoscopy, between January 2009 and December 2018. The primary outcome was incident dyslipidemia at follow‐up. Results: During the 10,324 person‐years of follow‐up, participants with persistent H. pylori infection had a higher incidence rate (130.5 per 1,000 person‐years) of dyslipidemia than those whose infections had been successfully controlled (98.1 per 1,000 person‐years). In a multivariable model adjusted for age, sex, waist circumference, smoking status, alcohol intake, and education level, the H. pylori eradication group was associated with a lower risk of dyslipidemia than the persistent group (HR, 0.85; 95% CI, 0.77–0.95; p = 0.004). The association persisted after further adjustment for baseline levels of low‐density and high‐density lipoprotein cholesterol (HR, 0.87; 95% CI, 0.79–0.97; p = 0.014). Conclusions: H. pylori infection may play a pathophysiologic role in the development of dyslipidemia, whereas H. pylori eradication might decrease the risk of dyslipidemia. [ABSTRACT FROM AUTHOR]
- Abstract: Copyright of Helicobacter is the property of Wiley-Blackwell and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)
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