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Large artery involvement in giant cell (temporal) arteritis.
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- Author(s): Klein RG; Hunder GG; Stanson AW; Sheps SG
- Source:
Annals of internal medicine [Ann Intern Med] 1975 Dec; Vol. 83 (6), pp. 806-12.
- Publication Type:
Case Reports; Journal Article
- Language:
English
- Additional Information
- Source:
Publisher: American College of Physicians--American Society of Internal Medicine Country of Publication: United States NLM ID: 0372351 Publication Model: Print Cited Medium: Print ISSN: 0003-4819 (Print) Linking ISSN: 00034819 NLM ISO Abbreviation: Ann Intern Med Subsets: MEDLINE
- Publication Information:
Publication: <2001->: Philadelphia, PA : American College of Physicians--American Society of Internal Medicine
Original Publication: Philadelphia [etc.] American College of Physicians.
- Subject Terms:
- Abstract:
Of 248 patients with giant cell arteritis, 34 had evidence that the disease affected the aorta or its major branches. Symptoms suggestive of large artery involvement were intermittent claudication of an extremity, paresthesias, and Raynaud's phenomenon. Physical findings included absent or decreased large artery pulses and bruits over large arteries. Four patients presented with decreased upper extremity pulses as the initial manifestation of their arteritis. Nine other patients under treatment for temporal arteritis or polymyalgia rheumatica first developed evidence of large artery involvement as corticosteroid therapy was tapered or discontinued. Angiography, performed in 10 patients, was helpful in indicating arteritis rather than atherosclerosis as the cause of large artery disease. Three patients died with aortic rupture, and, at autopsy, widespread giant cell arteritis was found. However, when corticosteroids were given in adequate doses, the response was favorable in most patients; intermittent claudication decreased and the pulses improved.
- Accession Number:
0 (Adrenal Cortex Hormones)
- Publication Date:
Date Created: 19751201 Date Completed: 19760213 Latest Revision: 20220419
- Publication Date:
20221208
- Accession Number:
10.7326/0003-4819-83-6-806
- Accession Number:
1200525
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